Book of Abstracts - New Frontiers 2022

Abstracts of poster presentations

but has no effects if either α - or β -subunits to IKs are functionally impaired. DHA could thus represent a novel therapeutic tool in LQT2 syndrome. This work was supported by a grant from the Hungarian National Research, Development, and Innovation Office (NKFIH K-128851).

Keywords: cardiac electrophysiology, long QT syndrome, impaired repolarization reserve, transgenic LQT rabbit models, polyunsaturated fatty acid, docosahexaenoic acid (DHA)

Funding: Hungarian National Research, Development, and Innovation Office (NKFIH K-128851)

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